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Parkin is a disease modifier in the mutant SOD1 mouse model of ALS. - Abstract - Europe PMC
Parkin is a disease modifier in the mutant SOD1 mouse model of ALS. - Abstract - Europe PMC

IJMS | Free Full-Text | Abnormal Upregulation of GPR17 Receptor Contributes to Oligodendrocyte Dysfunction in SOD1 G93A Mice
IJMS | Free Full-Text | Abnormal Upregulation of GPR17 Receptor Contributes to Oligodendrocyte Dysfunction in SOD1 G93A Mice

Figure 4 from Mutant superoxide dismutase 1 (SOD1), a cause of amyotrophic lateral sclerosis, disrupts the recruitment of SMN, the spinal muscular atrophy protein to nuclear Cajal bodies. | Semantic Scholar
Figure 4 from Mutant superoxide dismutase 1 (SOD1), a cause of amyotrophic lateral sclerosis, disrupts the recruitment of SMN, the spinal muscular atrophy protein to nuclear Cajal bodies. | Semantic Scholar

Amyotrophic Lateral Sclerosis disease - QPS Austria
Amyotrophic Lateral Sclerosis disease - QPS Austria

A New AAV10-U7-Mediated Gene Therapy Prolongs Survival and Restores Function in an ALS Mouse Model - ScienceDirect
A New AAV10-U7-Mediated Gene Therapy Prolongs Survival and Restores Function in an ALS Mouse Model - ScienceDirect

ALS-Linked SOD1 Mutants Enhance Neurite Outgrowth and Branching in Adult Motor Neurons
ALS-Linked SOD1 Mutants Enhance Neurite Outgrowth and Branching in Adult Motor Neurons

Figure 3.1 from The neuromuscular transmission of the SOD 1 ( G 93 A ) mouse model of Amyotrophic Lateral Sclerosis | Semantic Scholar
Figure 3.1 from The neuromuscular transmission of the SOD 1 ( G 93 A ) mouse model of Amyotrophic Lateral Sclerosis | Semantic Scholar

Generation and analysis of innovative genomically humanized knockin SOD1, TARDBP (TDP-43), and FUS mouse models - ScienceDirect
Generation and analysis of innovative genomically humanized knockin SOD1, TARDBP (TDP-43), and FUS mouse models - ScienceDirect

Transgenic mice expressing H46R/H48Q/H63G/H120G (Quad) mutant SOD1... | Download Scientific Diagram
Transgenic mice expressing H46R/H48Q/H63G/H120G (Quad) mutant SOD1... | Download Scientific Diagram

Frontiers | Neuroprotective Effects of Shenqi Fuzheng Injection in a Transgenic SOD1-G93A Mouse Model of Amyotrophic Lateral Sclerosis
Frontiers | Neuroprotective Effects of Shenqi Fuzheng Injection in a Transgenic SOD1-G93A Mouse Model of Amyotrophic Lateral Sclerosis

Therapeutic effects of immunization with mutant superoxide dismutase in mice models of amyotrophic lateral sclerosis | PNAS
Therapeutic effects of immunization with mutant superoxide dismutase in mice models of amyotrophic lateral sclerosis | PNAS

ALS-Linked SOD1 Mutant G85R Mediates Damage to Astrocytes and Promotes Rapidly Progressive Disease with SOD1-Containing Inclusions: Neuron
ALS-Linked SOD1 Mutant G85R Mediates Damage to Astrocytes and Promotes Rapidly Progressive Disease with SOD1-Containing Inclusions: Neuron

Guanabenz Treatment Accelerates Disease in a Mutant SOD1 Mouse Model of ALS | PLOS ONE
Guanabenz Treatment Accelerates Disease in a Mutant SOD1 Mouse Model of ALS | PLOS ONE

PDF] Wild-type SOD1 overexpression accelerates disease onset of a G85R SOD1 mouse. | Semantic Scholar
PDF] Wild-type SOD1 overexpression accelerates disease onset of a G85R SOD1 mouse. | Semantic Scholar

SOD1-positive aggregate accumulation in the CNS predicts slower disease progression and increased longevity in a mutant SOD1 mouse model of ALS | Scientific Reports
SOD1-positive aggregate accumulation in the CNS predicts slower disease progression and increased longevity in a mutant SOD1 mouse model of ALS | Scientific Reports

Disease progression in G93A-SOD1 transgenic mice is accelerated by... | Download Scientific Diagram
Disease progression in G93A-SOD1 transgenic mice is accelerated by... | Download Scientific Diagram

Overexpression of CCS in G93A-SOD1 mice leads to accelerated neurological deficits with severe mitochondrial pathology | PNAS
Overexpression of CCS in G93A-SOD1 mice leads to accelerated neurological deficits with severe mitochondrial pathology | PNAS

Neuroprotective effects of siRNAp11 in the SOD1-G93A mouse model of... | Download Scientific Diagram
Neuroprotective effects of siRNAp11 in the SOD1-G93A mouse model of... | Download Scientific Diagram

SOD1-G93A Mouse Models - QPS Neuropharmacology
SOD1-G93A Mouse Models - QPS Neuropharmacology

Frontiers | Skeletal Muscle-Restricted Expression of Human SOD1 in Transgenic Mice Causes a Fatal ALS-Like Syndrome
Frontiers | Skeletal Muscle-Restricted Expression of Human SOD1 in Transgenic Mice Causes a Fatal ALS-Like Syndrome

Parkin is a disease modifier in the mutant SOD1 mouse model of ALS | EMBO Molecular Medicine
Parkin is a disease modifier in the mutant SOD1 mouse model of ALS | EMBO Molecular Medicine

CuATSM improves motor function and extends survival but is not tolerated at a high dose in SOD1G93A mice with a C57BL/6 background | Scientific Reports
CuATSM improves motor function and extends survival but is not tolerated at a high dose in SOD1G93A mice with a C57BL/6 background | Scientific Reports

Frontiers | Human Motor Neurons With SOD1-G93A Mutation Generated From CRISPR/Cas9 Gene-Edited iPSCs Develop Pathological Features of Amyotrophic Lateral Sclerosis
Frontiers | Human Motor Neurons With SOD1-G93A Mutation Generated From CRISPR/Cas9 Gene-Edited iPSCs Develop Pathological Features of Amyotrophic Lateral Sclerosis

MicroRNA-206 Delays ALS Progression and Promotes Regeneration of Neuromuscular Synapses in Mice
MicroRNA-206 Delays ALS Progression and Promotes Regeneration of Neuromuscular Synapses in Mice

SOD1 aggregation in ALS mice shows simplistic test tube behavior | PNAS
SOD1 aggregation in ALS mice shows simplistic test tube behavior | PNAS